Vitreous is a clear gel that occupies the posterior segment of the eye which is made up of 98% water and 2% macromolecules (1, 2).   The outer cortex of vitreous which is made up of dense collagen (Type II) is firmly attached to internal limiting membrane (Type IV collagen). Vitreous is strongly attached at vitreous base, optic disc and to some extend on macula. As the age advances, gel undergoes liquefaction and attachment between vitreous and internal limiting membrane (ILM) weakens and it gets completely separated which is called posterior vitreous detachment(PVD), which usually begins in the perifoveal macula(3,4). PVD occurs in two phases– liquefaction followed by separation. The completion of vitreopapillary separation often characterized by the Weiss ring, is usually an acute and symptomatic event. Inadequate or incomplete vitreoretinal interface separation result in anomalous PVD with vitreomacular interface (VMI) anomalies. Anomalous PVD is defined as partial vitreous detachment with persistent attachment in the macular region featuring an anomalous strength of adhesion to one or more structures in the posterior pole, resulting in tractional deformation of retinal tissue (5).