Introduction: Hypertension is one of the few most attentive ailments throughout the globe because of its varied prevalence throughout the population irrespective of the region, religion and food habits, social and economic status. Low 25–hydroxyvitamin D [25(OH)D] levels are commonly found in patients with both Type 1 and Type 2 diabetes mellitus. Observational studies suggest that low 25(OH)D levels are associated with a higher risk of hypertension. PHPT is associated with increased risk of arterial hypertension. The present analysis was based on a cross–sectional study in the framework of comprehensive cardiovascular health examinations.

Materials & Methods: Statistical analysis was done using IBM SPSS Statistics 20 package. p–value of <0.05 is considered as statistically significant and p–value of <0.005 is considered as statistically highly significant. The results were averaged as (mean ± standard deviation) for each parameter subgroups separately for Hypertensives & Normotensives. Statistical analysis was done using IBM SPSS Statistics 20 package.  

Results: The values of Mean Systolic & Diastolic Blood Pressures of Hypertensives is found to be 153.76 mmHg and 86.32mmHg; and in Normotensives they are 137.27mmHg and 79.18mmHg. Mean Vitamin D in Hypertensives is 52.31ng/mL; and 31.43ng/mL in Normotensives. Similarly, the values of PTH in Hypertensives is 71.86ng/mL and 34.56ng/mL in Normotensives.

Discussion: In the present study, it is found that there is significant increase in Blood pressure in the individuals with decreased vitamin D levels. The levels of PTH were also found to be significantly less in the individuals with increased blood pressure. Dietary intake of sodium and increased activity of the RAS are known to be linked with increased blood pressure; salt restriction and inhibition of RAS activity is found to be linked with reduced blood pressure. Changes in systemic calcium metabolism are thought to play an important role in the regulation of blood pressure. One hypothesis for this link implicates parathyroid hormone (PTH). Serum calcium level is tightly regulated by PTH in a classic negative feedback system