Volume : IV, Issue : III, March - 2015

Abstract :

 Carbon monoxide (CO) poisoning is a significant cause of morbidity and mortality in our country, especially in winter, 

because of poorly functioning heating systems. We present a case with severe acute carbon monoxide poisonıng. The 
30–year–old male was previously healthy and non–smoker. He was found lying unconscious on the floor. Although his father 
smelled a pungent odor and felt headache, dizziness, agitation, and dyspnea after entering the room, he had realized 
that he was apneic and than he gave her mouth–to–mouth respiration for 10 minutes before eathing resumed. He was 
taken to a local hospital and received oxygen via nasal cannula (10 L/minute) within 30 minutes. First cranial tomography 
(CT) findings were unremarkable other than ain edema. He was admitted to intensive care unit. Glascow score was 4. 
His arterial blood gas (ABG) sample analysis revealed metabolic acidosis and hipoxemia. Carboxyhemoglobin (COHb) level 
was 59.4 % and electrocardiography showed a mild ST– segment depression over anterior leads, suggestive of myocardial 
ischemia. HBO2 therapy was immediately initiated within 4 hours after exposure to CO in a multiplace chamber. HBO2 
therapy was withheld after completing nine session. His symptoms improved after first HBO2 therapy and COHb level was 
23 %. He was discharged on day of 5 and a normal follow–up five weeks after discharge. It has been shown that HBO2 
therapy has provided prominent improvement in the early and late effects of CO poisoning and this improvement is more 
quick and more effective in acute phase.

Article: Download PDF    DOI : https://www.doi.org/10.36106/paripex  

Cite This Article:

, PARIPEX-INDIAN JOURNAL OF RESEARCH : Volume-2 | Issue-3 | March-2013


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